SECHRIST 3200 Hyperbaric Chamber

In mint condition. For more questions, feel free to email me at or check directly at Our price US$10,690


30 days money back guarantee. 1 year warranty for parts in future.

SeChrist 3200-a    SeChrist 3200-b

· Gurney and Bed Included
· Brand new mattress and pillow
5762 cycles. Includes Stryker gurney and all hoses and hardware. 2003 model.
6 month warranty for parts & labor. We have 5 unit in stock.

The Sechrist 3200 is a pneumatically controlled system, which features a spacious 32 inch inside diameter acrylic cylinder for increased patient mobility and comfort. Important capabilities such as ventilation support and intravenous infusion can also be provided. Transcutaneous and other monitoring is possible via electrical ports and pass-throughs. Additional features include a hyperbaric gurney for easy patient transport, an intercom system for two-way private conversation and an auxiliary audio-input for patient entertainment.

What is Hyperbaric Oxygen Therapy?
Hyperbaric Oxygen Therapy (HBOT) is a medical treatment in which the patient is entirely enclosed in a pressure chamber breathing 100% pure oxygen (O2) at greater than one atmosphere pressure. Air contains nearly 21% oxygen, and approximately 78% nitrogen. In hyperbaric oxygen therapy (HBO), the oxygen percentage breathed by the patient is nearly or actually 100%, almost five times more than in air. The pressure of the oxygen breathed by the patient in a hyperbaric oxygen chamber is usually more than 1.5 times (and can be as much as 3 times) greater than atmospheric pressure. HBOT can deliver nearly 15 times as much oxygen as there is in air at normal pressure.

What are the clinically accepted indications for Hyperbaric Oxygen Therapy?
HBOT has been proven effective for many medical conditions, and as a result the Undersea and Hyperbaric Medicine Society, one of the premier research institutes, has approved following indications:
Air or Gas Embolism
Carbon Monoxide Poisoning Carbon Monoxide Poisoning Complicated by Cyanide Poisoning
Clostridal Myositis and Myonecrosis (Gas Gangrene)
Crush Injury, Compartment Syndrome, and other Acute Traumatic Ischemias
Decompression Sickness
Enhancement of Healing in Selected Problem Wounds; Arterial Insufficiencies; Central Retinal Artery Occlusion
Severe Anemia
Intracranial Abscess
Necrotizing Soft Tissue Infections
Refractory Osteomyelitis
Delayed Radiation Injury (Soft Tissue and Bony Necrosis)
Compromised Skin Grafts & Flaps
Acute Thermal Burn Injury
In addition, Medicare coverage determinations will reimburse in the U.S. for the following conditions:

Diabetic wounds of the lower extremities in patients who meet the following three criteria:

Patient has type I or type II diabetes and has a lower extremity wound that is due to diabetes;
Patient has a wound classified as Wagner grade III or higher; and
Patient has failed an adequate course of standard wound therapy.

What are the physiological mechanisms of action caused by HBOT that make it work?
Hyperoxygenation: HBOT physically dissolves extra oxygen into the blood plasma, which is then delivered to the tissues. Breathing pure oxygen at two to three times normal pressure delivers 10-15 times as much physically dissolved oxygen to tissues. This can increase the tissue oxygen in compromised tissues to greater-than-normal values. Hyperoxygenation has been demonstrated to induce formation of new capillaries in ischemic or poorly perfused wounds. Therefore, it is useful in the treatment of ischemic based compromised wounds, flaps and grafts. It is also helpful in some infections by allowing white cell (leukocytic) activity to resume function.

Mechanical Effect of Increased Pressure: Any gas in the body will decrease in volume as the pressure on it increases. With a threefold increase in pressure, a bubble trapped in the body is reduced by two-thirds. Thus, reduction in gas volume resolves air embolism and decompression illness when the diagnosis to treatment is done in a timely manner.

Vasoconstriction: High-pressure oxygen causes constriction of the blood vessels in normal tissues without creating hypoxia. It does not cause constriction in previously oxygen-deprived tissues. The vasoconstriction decreases edema which is helpful in the treatment of burns, crush injuries, compartment syndromes and other acute traumatic ischemias. Even though the blood flow that contributes to edema is reduced, oxygen delivery to the tissues is maintained through the hyperoxygenation effect.

Antimicrobial Activity: HBOT inhibits alpha toxin production as seen in anaerobic infections such as clostridium perfringens (gas gangrene). The most common cause of gas gangrene is clostridium perfringens; however, there are several gas producing organisms (aerobic and anaerobic) that require surgical debridement initially. It also enhances the white cell killing activity which provides an excellent adjuvant to I.V. antibiotic and local wound care.

Mass Action of Gases: The flooding of the body with any one gas tends to “wash out” others. This action occurs more rapidly under pressure than under ordinary conditions, and makes HBOT an indicated treatment for decompression sickness.

Reduction of Reperfusion Injury: Following an ischemic interval, indirect injury occurs, which is mediated by the inappropriate activation of leukocytes. HBOT prevents such activation. Adherence of white blood cells to capillary walls is markedly reduced, thus mitigating the “no reflow” phenomenon. This is why HBOT therapy is indicated in carbon monoxide poisoning and is considered the treatment of choice.

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